心肌损伤是由于心脏I/R造成，长期I/R损伤可能引发DNA损伤、细胞功能障碍和细胞死亡，可导致急性冠脉综合征和心肌梗死,严重威胁病人的生命健康。柠檬酸盐（柠檬酸锶Strontium Citrate）是一种重要代谢产物，不仅可以作为胆固醇与细胞质合成脂肪酸的碳源，还可以促进线粒体能量代谢，使用其预处理可以有效降低心肌细胞凋亡, 有研究表明，柠檬酸盐（柠檬酸锶Strontium Citrate）具有抑制新鲜血液凝固的作用，在临床采血中被用作抗凝血剂，并有报道指出其可以起保护心肌的作用，有效抑制心肌细胞凋亡;在心肌细胞研究中，柠檬酸盐（柠檬酸锶Strontium Citrate）展现出独特作用。当心肌细胞经历缺氧/复氧过程时，往往会遭受严重损伤。而柠檬酸锶（Strontium Citrate）能通过多种机制发挥保护作用。它可以调节细胞内离子平衡，减少因缺氧/复氧导致的钙超载现象，降低心肌细胞的兴奋性，维持其正常的电生理活动。同时，柠檬酸锶（Strontium Citrate）可能具有抗氧化作用，清除自由基，减轻氧化应激对心肌细胞的损害。此外，它还可能参与细胞信号传导通路的调节，促进心肌细胞的存活和修复，为防治心肌缺血再灌注损伤提供了新的思路和方向

柠檬酸锶Strontium Citrate对缺氧/复氧诱导的心肌细胞损伤的影响

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Effect of Strontium Citrate on hypoxia/reoxygenation-induced cardiomyocyte injury

Myocardial injury is caused by cardiac I/R, and long-term I/R damage may lead to DNA damage, cell dysfunction and cell death, which can lead to acute coronary syndrome and myocardial infarction, which seriously threatens the life and health of patients. Citrate (Strontium Citrate) is an important metabolite, which can not only be used as a carbon source for the synthesis of fatty acids from cholesterol and cytoplasm, but also promote mitochondrial energy metabolism. Studies have shown that citrate (Strontium Citrate) has the effect of inhibiting the clotting of fresh blood, and is used as an anticoagulant in clinical blood collection. It has also been reported that it can play a protective role in myocardium and effectively inhibit cardiomyocyte apoptosis; In cardiomyocyte research, citrate (Strontium Citrate) exhibits a unique role. When cardiomyocytes undergo a hypoxia/reoxygenation process, they tend to suffer severe damage. Strontium Citrate, on the other hand, exerts a protective effect through a variety of mechanisms. It can regulate intracellular ion balance, reduce calcium overload caused by hypoxia/reoxygenation, reduce the excitability of cardiomyocytes, and maintain their normal electrophysiological activities. At the same time, Strontium Citrate may have antioxidant effects, scavenge free radicals, and reduce oxidative stress damage to cardiomyocytes. In addition, it may also participate in the regulation of cell signaling pathways, promote the survival and repair of cardiomyocytes, and provide new ideas and directions for the prevention and treatment of myocardial ischemia-reperfusion injury

Effect of Strontium Citrate on hypoxia/reoxygenation-induced cardiomyocyte injury

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