铁通过控制的稳定性和翻译来调节调节转铁蛋白和铁蛋白的水平。研究证明由于铁缺乏导致的转铁蛋白基因表达的增加是通过提高转录水平来实现的。铁对铁蛋白基因表达的调控与转铁蛋白基因表达控制不同当铁存在时它能与铁反应要素结合导致暴露翻译起始位点‚使得细胞很快合成铁蛋白而且铁含量越高铁蛋白基因表达就越快；当铁的供给不足时，起始位点被铁反应要素覆盖铁蛋白合成快速停止。缺铁或者铁的利用不良将导致氧的运输、贮存、二氧化碳的运输以及氧化还原等代谢过程紊乱影响生长发育甚至发生贫血等各种疾病。

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抗坏血酸亚铁Ferrous Ascorbate、赖氨酸甘氨酸镁Magnesium Lysinate Glycinate、甘氨酸谷氨酰胺镁Magnesium Glycinate Glutamine、柠檬酸苹果酸镁Magnesium Citrate Malate、柠檬酸锶Strontium Citrate、柠檬酸锰Manganese Citrate、柠檬酸铜Copper Citrate、天门冬氨酸锂Lithium Aspartate、抗坏血酸锰Manganese Ascorbate、牛磺酸硒Selenium Taurate。

The role of iron in human cells

Iron regulates the levels of regulatory transferrin and ferritin through controlled stability and translation. The study demonstrated that the increased expression of the transferrin gene due to iron deficiency is achieved by increasing the level of transcription. The regulation of ferritin gene expression by iron is different from the control of transferrin gene expression. In the presence of iron, it can bind to the iron-reactive elements, resulting in exposure to the translation initiation site ‚, which enables cells to synthesize ferritin quickly and the higher the iron content, the faster the ferritin gene expression; When the supply of iron is insufficient, the initiation site is coated with iron reactive elements and ferritin synthesis stops rapidly. Iron deficiency or poor use of iron will lead to oxygen transport, storage, carbon dioxide transport and REDOX and other metabolic processes disorders affect growth and development and even anemia and other diseases.

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Ferrous Ascorbate, Magnesium Lysinate Glycinate, Magnesium lysinate Glycinate Glutamine, Magnesium citrate malate Citrate Malate, Strontium Citrate, Manganese Citrate, Copper Citrate, Lithium Aspartate, Manganese ascorbate Ascorbate, Selenium Taurate.