糖尿病是一种常见病、多发病，以其高发病率、高致死率严重影响人们的生活质量而被广泛关注。随着病情的发展，能诱发多种严重的并发症，其并发症主要分两大类，即糖尿病大血管并发症如糖尿病大血管病变、动脉粥样硬化；糖尿病微血管并发症如糖尿病心肌病，糖尿病肾脏病，糖尿病视网膜病变等等，很多报道指出，氧化应激损伤对糖尿病以及其并发症的发生与发展负有不可推卸的责任。众所周知，糖尿病能引起机体血糖增高，代谢系统紊乱。这会导致组织器官炎症反应，氧化应激水平增高，氧化应激产物活性氧自由基(ROS)、活性氮自由基(RNS)产生增多，在组织器官中的累积也相应增多。它会攻击机体的组织器官、血管内皮细胞，产生一系列的损伤反应，例如炎症、纤维化、凋亡等等。流行病学报道指出，糖尿病患者血清锌离子含量降低。而外源性的补锌（苏氨酸锌Zinc Threoninate、柠檬酸锌Zinc Citrate、抗坏血酸锌Zinc Ascorbate），能使血清中锌离子含量增高，对糖尿病引起的血管损伤有预防和保护作用，而产生抗氧化作用。

锌（苏氨酸锌Zinc Threoninate、柠檬酸锌Zinc Citrate、抗坏血酸锌Zinc Ascorbate）对糖尿病血管损伤的预防和保护作用

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抗坏血酸锰Manganese Ascorbate、抗坏血酸亚铁Ferrous Ascorbate、赖氨酸甘氨酸镁Magnesium Lysinate Glycinate、甘氨酸谷氨酰胺镁Magnesium Glycinate Glutamine、柠檬酸苹果酸镁Magnesium Citrate Malate、柠檬酸锶Strontium Citrate、柠檬酸锰Manganese Citrate、柠檬酸铜Copper Citrate、天门冬氨酸锂Lithium Aspartate、牛磺酸硒Selenium Taurate.

The preventive and protective effects of zinc (zinc threoninate, zinc citrate, zinc ascorbate) on diabetic vascular damage

Diabetes is a common and frequently occurring disease, which has attracted wide attention due to its high incidence and high mortality rate, which seriously affects people's quality of life. As the disease progresses, it can induce a variety of serious complications, which are mainly divided into two categories, namely, diabetic macrovascular complications such as diabetic macrovascular disease and atherosclerosis; diabetic microvascular complications such as diabetic cardiomyopathy, diabetic nephropathy, diabetic retinopathy, etc. Many reports point out that oxidative stress damage has an unshirkable responsibility for the occurrence and development of diabetes and its complications. It is well known that diabetes can cause increased blood sugar and metabolic disorders in the body. This will lead to inflammatory reactions in tissues and organs, increased levels of oxidative stress, increased production of oxidative stress products, reactive oxygen free radicals (ROS) and reactive nitrogen free radicals (RNS), and correspondingly increased accumulation in tissues and organs. It will attack the body's tissues, organs, and vascular endothelial cells, producing a series of damage reactions, such as inflammation, fibrosis, apoptosis, etc. Epidemiological reports indicate that the serum zinc ion content of diabetic patients is reduced. Exogenous zinc supplementation (zinc threoninate, zinc citrate, zinc ascorbate) can increase the zinc ion content in serum, prevent and protect against vascular damage caused by diabetes, and produce antioxidant effects.

The preventive and protective effects of zinc (zinc threoninate, zinc citrate, zinc ascorbate) on diabetic vascular damage

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Manganese Ascorbate, Ferrous Ascorbate, Magnesium Lysinate Glycinate, Magnesium Glycinate Glutamine, Magnesium Citrate Malate, Strontium Citrate, Manganese Citrate, Copper Citrate, Lithium Aspartate, Selenium Taurate.